Written by a Medical Acupuncture for Veterinarians course graduate. Signed release obtained from client/author. 4376
Nezumi, 16yr Mn Chi presented for caudal cervical lesion treatment complicated with challenges of congenital MPL, hock hyperextension, elbow dysplasia and temperament. He has had acupuncture before where the owner (O) was instructed to hold him down since he would bite.
History and Presentation:
Nezumi’s problems started acutely 3 weeks prior when he started stumbling with his right front leg. Nezumi has history of patella luxations and elbow arthritis for years. There is also a history of suspected atopy as he licks his front feet however this is confined to the medial carpus (left more then right). There is no known trauma. He has been owned since a puppy.
MEDS; CBD oil, dasequin chews DIET: Homecooked, chicken based
Initial physical exam: **abnormalities only**
- GA: BARH, caution near head and elbows!; Pain Score 8/10
- INTEG: Intense lick staining medial carpi (Left>Right) EENT : Mild tartar, oligodontia,
- MS: amb x4, Double engine gait with paresis of right thoracic limb (RTL) and ataxic wide based PL stance. Hyperextended hocks with bilateral ¾ medial luxated patella (MPL). Right (R) stifle thickening. Bilateral elbow thickening with abduction (L>R) and L elbow heat. Pain elicited on C6 palpation. Mild focal pain on thoracolumbar (TL) junction.
- NEURO: Absent conscious proprioception RTL and delayed LTL, Normal PL. Intact pain sensation x4. Intact panniculus. Normal to hyper patellar reflex.
- Myofascial palpation: Acute pain in caudal cervical region however compensatory muscle strain/trigger points were severe of neck, thorax, lumbar, semitendinosus and crus.
- Right lateralized C6-T2 lesion: suspect intervertebral disc disease (IVDD) vs other
- Caudal cervical acute pain with proprioceptive deficits of both TL and the PL ataxia
- Suspected chronic cervical nerve radiculopathy (C7-T1) vs less likely atopy
- Chronic licking of the medial carpi rather than all foot pads
- Suspected chronic thoracolumbar (TL) lesion r/o IVDD
- Focal TL pain with intact panniculus and HL proprioception
- Elbow dysplasia with osteoarthritis (OA). Severe on left/moderate on right.
- Bilateral medial luxated patellas (MPL) with hyperextension of hocks and R stifle OA
- Myofascial restriction and trigger points of PL, lumbar (marked), thoracic, cervical, triceps regions
- Suspected cushing disease and dental disease
Over time, congenital MPL caused more strain of lumbar core muscles and the TL region due to stabilization for gaits. This results in myofascial pain syndrome (MPS) of the lumbar, thorax, and front limbs (8, 10, 13). As time went on, the progressive arthritis of the elbows causes compensatory elbow abduction. While the original cause is unknown, I suspect that the progressive elbow abduction as well as afore described compensations exacerbated the chronic cervical nerve radiculopathy. The left radiculopathy caused weight shifting to the R side resulting in more trigger points of the RTL and lumbar area. The acute C6-T1 lesion then created more muscle strain of the neck muscles and further exacerbated MPS of the hind legs.
**Temperament prevented treatment near head even though he loved his frozen baby food
**When needling was not tolerated, massage or LED wavelength laser treatment was applied
- Autonomic: GV14, Bai Hui, ST 36, LR 3, BL 40
- Increase parasympathetic tone; Master points for lumbar area (13, 5)
- Peripheral: BL 10, BL 13, BL 14, BL 15, LU 1, LU 7, TH 10, TH 5, LI 4
- Distal sites for neural stimulation of LTL
- Local: GB 21, BL 13, BL 14, BL 15, SI 11, SI 12, LI 11, BL 18, BL 20, BL 22, BL 25, ST 34, BL 40
- Caudal cervical pain, shoulder pain, elbow pain, TL/lumbar pain, stifle pain (6)
- EA: GV 14 to LI 15; GB 21 to BL 15 **temperament prevented EA stimulation of leg itself
- Cervical pain control and neural stimulation (3, 4, 5, 9, 2, 14)
- EA: BL 20-BL 25
- Lumbar pain control (3, 4, 6, 14)
- Dry needling was used for myofascial trigger points found in the lumbar muscles (10, 13)
- Metacam PO q24hr x 7d, Gabapentin 5mg/kg PO q12hr x14d
- O instructed weight shifting on stairs, regular walking, red laser pointer treatment to mid-scapular area and stifle area. Gentle walking and to encourage LTL foot placement. (1, 2, 15)
- Bi-weekly sessions for 2 weeks then reduced to weekly maintenance. At home sessions consisted of 20-30min acupuncture needling, 10min EA stimulation and 10-15min massages.
- Needles were Sierin 0.16g x 15mm and 0.2g x 15mm for EA; carbo 0.2 x 13mm
Caudal cervical lesions are commonly encountered in veterinary medicine with the majority related to disc rupture (5). Multiple studies have shown great, if not better benefit of electroacupuncture and photomedicine to these neuronal injuries (3, 5). Ideally photomedicine can be used to improve axonal healing however neural growth can still occur for motor and sensory function using stimulation with acupuncture, EA and controlled exercise (14, 6, 1, 2, 11).
Nezumi showed progressive improvement and within 2weeks, he was ambulating well without assistance although foot placement when standing took closer to 4 weeks for recovery.
Prior to curacore training, medical recommendations included medical treatment and 4-6 weeks of strict rest. This rest reduces fibroblast populations in joints, ECM, and muscle as well as not stimulating axonal growth and repair (1, 2, 7, 12, 15).
While Nezumi’s temperament created a challenge, with patience, we were able to elicit a change and return to almost normal function. Video is provided for pre treatment, 3 weeks post and again 4weeks post treatment. This experience was a win-win-win situation. Nezumi’s pain level was quickly controlled and function returned, the owner-pet bond was strengthened and I could see and feel benefits of my work. Medical acupuncture is a tangible way to work with neurophysiology and anatomy of a patient. It is an amazing experience. This also created a different level of communication with the owner regarding neuro, immune and vascular benefits that strengthened the trust level between us.
Nezumi has been doing well however about 3 months after initiation, his clinical signs acutely worsened of the same leg and I worried that either a new disc ruptured or this was a sign of other pathology (neoplasia). We increased his treatments and fortunately, function progressively returned again after 3-4 weeks. The most exciting news is that the past few weeks, Nezumi actually crawled into my lap with licks of affection and let me do most of the treatments… except, of course, with the head.
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